Nanolive Joins as Official Supporter of World Mitochondria Society Congress 2025
The World Mitochondria Society is proud to announce its partnership with Nanolive for the 16th World Mitochondria Congress, to be held from the 22th to 24th of October in Berlin, Germany.
Nanolive, a Swiss biotech company based in Lausanne, is at the forefront of label-free live-cell imaging. Their cutting-edge technology allows researchers to observe living cells in 3D, in real-time, without using dyes or fluorescent markers, a major breakthrough for mitochondrial and cellular research.
Nanolive’s unique imaging systems combine quantitative data acquisition (such as dry mass and refractive index) with dynamic live-cell analysis, providing unprecedented insights into cellular processes such as mitochondrial dynamics, mitophagy, apoptosis, and bioenergetic responses.
Thanks to its AI-powered analytics, Nanolive enables high-throughput screening and detailed time-lapse analyses, empowering researchers to monitor cellular health, metabolic shifts, and drug responses with exceptional precision and without altering cell physiology. This includes the Smart Mitochondrial AssayLIVE, the world’s first fully automated solution for label-free, quantitative assessment of mitochondrial dynamics over time.
At the 2025 Congress, Nanolive will present how its label-free imaging platforms are being used to explore mitochondrial function in health and disease, and how this approach can accelerate the discovery of new therapies targeting mitochondrial dysfunction.
We are excited to count Nanolive among our partners and look forward to their contribution to the advancement of mitochondrial science.
For more information about Nanolive: www.nanolive.com
Lucid Scientific Joins as Official Supporter of World Mitochondria Society Congress 2025
The World Mitochondria Society officially welcomes Lucid Scientific as a supporter of the 16th World Mitochondria Society Congress, taking place from October 22–24, 2025, in Berlin, Germany.
Lucid Scientific, a biotechnology company based in Atlanta, Georgia, is renowned for its innovative solutions in cell culture research. Their flagship product, Resipher, is the world's first real-time, non-invasive cell culture monitor that measures oxygen consumption directly within standard incubator environments.
The Resipher system empowers researchers to precisely measure oxygen consumption directly in standard microplates. Its patented dynamic optical oxygen sensors provide high sensitivity without disturbing cells. Resipher's compact design allows it to sit directly in incubators, and its web-based software streams real-time data to the cloud, enabling researchers to monitor experiments remotely via computer, smartphone, or tablet.
At the 2025 Congress, Lucid Scientific will showcase how Resipher's capabilities can advance mitochondrial research by providing continuous, high-resolution data on cellular respiration. This aligns with the Congress's focus on translating the latest mitochondrial research into tangible medical treatments.
For more information about Lucid Scientific and the Resipher system : lucidsci.com.
Targeting Mitochondria 2025 Best Image Submissions
The flame within
By Parnasree Mahapatra, Ph.D student, Ion channel lab, School of Biological Sciences, National Institute of Science Education and Research, Jatni, Khurdah, India
Study Context : Super resolution image of mitochondria labelled with Mito tracker red and transfected with TRPV4 MTS DNA construct in Saos-2 cell
Lighting Up Mitochondrial Stress: SNX9 Dynamics in Parkinson’s disease
By Jimna Mohamed Ameer, PhD student, Inter University Centre for Biomedical Research and Super Speciality Hospital, Mahatma Gandhi University, Kottayam, Kerala, India
Study Context: MPP⁺-induced mitochondrial dysfunction in differentiated SH-SY5Y cells models early events in Parkinson’s disease. This neurotoxic stress causes mitochondrial fragmentation and disrupts endolysosomal functions. Our results reveal that Sorting Nexin 9 (SNX9), a membrane remodelling protein involved in endocytosis, is recruited to dysfunctional mitochondria following MPP⁺ treatment. This suggests a potential role for SNX9 in mitochondrial-derived vesicle (MDV) formation or mitochondrial quality control, providing novel insights into mitochondrial signalling and vesicular trafficking during neurodegenerative stress responses.
Glow and behold: Mitochondria on the move
By Phua Qian Hua, Institute of Molecular and Cell Biology, Agency for Science, Technology and Research (ASTAR),Singapore, National University of Singapore
Study Context : Confocal microscopy image taken after mitochondrial transplantation from healthy iPSC-derived mitochondria (labeled with MitoTracker Green) into diseased endothelial cells with dysfunctional endogenous mitochondria (labeled with MitoTracker Red).
Human cardiomyocytes
By Andrea Elia, Alzheimer's Center at Temple (ACT), Department of Neural Sciences, Lewis Katz School of Medicine, Temple University, Philadelphia
Study Context : Human cardiomyocytes serve as a powerful model to elucidate the biological impact of Alzheimer’s disease-associated amyloid-β (Aβ) pathology on cardiac function, enabling direct investigation into the cellular mechanisms driving Aβ-induced cardiac dysfunction.Aβ40 oligomers impair mitochondrial architecture and destabilize cytochrome c integrity in human cardiomyocytes, indicating a potential mechanism of amyloid-induced cardiac dysfunction.
Glowing and Growing Mind: Mitochondria in Early Neural Development
By Sundas Arshad, Luxembourg centre for Systems Biomedicine, University of Luxembourg
Study Context : This image is part of a study investigating mitochondrial remodeling during early neuronal differentiation. LUHMES cells, which are human embryonic neuronal precursor cells, have been utilized in several studies to investigate the PD and underlying mechanisms associated with it. Mitochondria were stained using TOM20, a marker of the outer mitochondrial membrane, to visualize their distribution and morphology. The goal of our study is to investigate the crosstalk between Ca²⁺ signaling and mitochondrial dynamics in dopaminergic neurons by combining live cell imaging and immunostaining techniques.
Bumpy mitochondrial track: a roadway to disease
By Ritoprova Sen, Integrated Ph.D. student of Biological Sciences, Jawaharlal Nehru Centre for Advanced Scientific Research, Bengaluru, India
Study Context : Our study aims to investigate mitochondrial damage and quality control in the context of mitochondrial diseases, particularly in cardiomyocytes. One such mitochondrial disease associated mutant causes elongated mitochondria as observed in AC16 human cardiomyocyte cell line. This is a projected fluorescent microscopy image depicting an AC16 human cardiomyocyte cell expressing a dominant negative disease mutant exhibiting hyper-elongated mitochondria. The outer mitochondrial membrane has been labelled with a construct expressing OMP25 (labelled in magenta) while the green denotes COXIV, an inner mitochondrial membrane protein which is a part of complex IV of the electron transport chain. The mutant manifests as elongated mitochondria, interrupted by bumps in the form of swollen bulbs as seen in magenta. Interestingly these swollen bulbs do not costain with COXIV in green. Whether such mitobulbs cause localized mitochondrial damage and consequently activation of mitochondrial quality control is currently an active area of investigation in the lab.
Galacto’Mitochondria in Cancer universe
By Lea Di Mascio, Research Engineer in molecular biology, Team 3 “ Metabolism, Cancer and Immune Response” at the Molecular Medicine Mediterranean Center (C3M) of Nice
Study Context : Our team is studying how mitophagy is involved in lung cancer.
Mitoverse: Multiverse of Mitochondria
By Pratiti Rout, PhD Scholar Justus-Liebig-Universität Gießen and doing part of her doctoral research at Philipps-Universität Marburg
Description: The image showcases how in a single tissue (seminal vesicle of Drosophila male reproductive system) mitochondria can adapt to such diverse architectures (interconnected networks- * in yellow, fragmented and globular shapes - * in white). Phalloidin marks actin in red, DAPI stains the nuclei in blue and mitochondria in the muscles of seminal vesicle is visualised in green with Mef2-Gal4 driven UAS-mitoGFP. MEF is muscle-specific transcription factor.
Context of the study: Mitochondrial dynamics is crucial for muscle function. In this study, we explore the role of mitochondrial architecture in muscle development and function using Drosophila as a model organism. In our attempt to observe differences in mitochondria morphology in different muscle types found in Drosophila male reproductive tissues, we aquired this image using Leica Stellaris confocal microscope. Accessory glands like seminal vesicles are usually surrounded by multinucleated striated muscles whereas testes has smooth-like muscles. Surprisingly, we observed different shapes of mitochondria in muscles of one single tissue (seminal vesicle) compelling us to reflect on the idea of 'multiverse of mitochondria'.
Submit your Image and win a Free Registration
Have a memorable mitochondria image?
Submit your image for a chance to win free in-person or virtual registration to the 2025 congress.
We also welcome original artwork or drawings related to mitochondria, whether it's about life, energy, or dynamics.
Image Submission Guidelines:
To enter the contest, please submit the following information in a Word document:
- Your name
- Your complete affiliation
- A picture of yourself (optional)
- Your mitochondria image, including: A title/ A description of the image/ The context of the study
How to Enter?
Send your submissions to: mitochondria[at]wms-site.com.
Share your vision of mitochondria and stand out!
Mitochondria & Organelle Crosstalk - Rethinking Organelle Crosstalk: Mitochondrial-Derived Vesicles in Peroxisome Biogenesis Presented by Dr. Ayumu Sugiura
At the heart of cellular metabolism, mitochondria and peroxisomes play tightly interconnected roles in lipid regulation, redox homeostasis, and energy dynamics. While direct contacts between these organelles have long been observed, the mechanisms underlying their communication and biological significance are only beginning to emerge.
In an insightful presentation, Dr. Ayumu Sugiura of Juntendo University, Japan, introduces a compelling hypothesis: mitochondrial-derived vesicles (MDVs) may serve as essential mediators in peroxisome biogenesis. These vesicles, generated by mitochondria in response to cellular cues, could carry lipids, enzymes, or signaling molecules critical for initiating or modulating peroxisomal function.
“Mitochondrial-derived vesicles may provide a missing mechanistic link in understanding how mitochondria influence peroxisome formation and specialization,” says Dr. Sugiura.
His talk emphasized that this vesicular communication is not a byproduct of stress or degradation but a targeted and regulated form of inter-organelle signaling, reflecting a deeper evolutionary connection.
Understanding MDVs and their role in peroxisome biology may open new avenues in treating metabolic disorders, neurodegenerative diseases, and inherited mitochondrial syndromes, where organelle cooperation is often impaired.
This new perspective encourages scientists to rethink organelle crosstalk not as static interactions but as dynamic exchanges of molecular information, and places MDVs at the center of this emerging dialogue.
Book Your Hotel at Reduced Rate
The 16th World Congress on Targeting Mitochondria on October 22-24, 2025, at
DoubleTree by Hilton Berlin Ku'damm
Los-Angeles-Platz 1
10789 Berlin, Germany
Book your room with a special rate
The WMS arranged a deal for all attendees, a special reduced rate for all rooms booked at the DoubleTree by Hilton.
King Guest Room: €209 per night
- Includes complimentary breakfast
- Surcharge for double occupancy: €20 per day
- City Tax not included – 7.5% ( with the new German law, all stays in hotels are taxed for personal & professional use)
How to Reach DoubleTree by Hilton:
1. From Berlin Tegel Airport (TXL):
- By Taxi: Taxis are readily available outside the terminal. The journey takes approximately 15-20 minutes, depending on traffic.
- By Public Transport: Take the X9 bus towards Zoologischer Garten. The hotel is a short walk from there.
2. From Berlin Schönefeld Airport (SXF):
- By Taxi: Taxis are readily available outside the terminal. The journey takes approximately 30-40 minutes, depending on traffic.
- By Public Transport: Take the regional train RE7 or RB14 to Zoologischer Garten, then a 10-minute walk.
3. By Train from Berlin Hauptbahnhof:
- Take S-Bahn lines S3, S5, S7, or S9 to Zoologischer Garten, then a brief walk.
4. By Car:
- The hotel is near the A100 city motorway.
- Parking is available at the hotel for a fee.
5. Public Transportation:
- U2 subway line stops at U-Bahnhof Zoologischer Garten.
- Numerous bus lines stop nearby.
World Mitochondria Society
16th Targeting Mitochondria 2025 Congress
October 22-25, 2025 - Berlin, Germany
www.wms-site.com
Targeting Mitochondrial Pyruvate Carrier: Impact on Future Metabolic Therapies
Prof. Edmund Kunji from the University of Cambridge will give a major talk entitled Targeting mitochondrial pyruvate carrier: impact on future metabolic therapies, during the Targeting Mitochondria 2025 Congress, which will be held on October 22-24, in Berlin, Germany.
About Prof. Kunji's talk:
Fifty years after the mitochondrial pyruvate carrier (MPC) was first identified, researchers have now resolved its molecular structure and mechanism of action. In a landmark study published in Science Advances, Sichrovsky et al. (2025) unveiled how this critical mitochondrial complex mediates pyruvate transport and how its inhibition could be leveraged for therapeutic purposes in cancer, metabolic disorders, and more.
About his outstanding study:
Major Discoveries of the Study by Prof. Edmund Kunji and his teams
Molecular Structure of MPC:
The authors used cryo-electron microscopy to capture the architecture of the human MPC complex. They discovered that MPC forms a heterodimeric transport unit (MPC1/MPC2), creating a selective channel that guides pyruvate across the inner mitochondrial membrane.
Mechanism of Transport and Inhibition:
The study revealed how small-molecule inhibitors bind to the MPC complex and block its function, offering a blueprint for drug development. Structural analysis pinpointed specific binding sites that explain both transport dynamics and inhibition sensitivity.
Conserved Functionality:
Evolutionary conservation of the MPC mechanism across species (including yeast and human) underscores its universal biological role in cellular energy homeostasis.
Therapeutic Implications
Cancer:
Some tumors overexpress MPC to fuel high mitochondrial activity. MPC inhibitors could starve these cells of essential metabolites, selectively disrupting their growth.
Metabolic Diseases:
In conditions like non-alcoholic fatty liver disease (NAFLD), blocking MPC forces hepatocytes to burn fat instead of relying on glucose, leading to reduced liver fat accumulation.
Regenerative Medicine & Hair Growth:
MPC inhibition has been shown to stimulate lactate production, which may promote hair follicle cell activation, opening potential new treatments for alopecia.
Mitochondrial Dysfunction & Neurodegeneration:
Targeting MPC may allow modulation of energy metabolism in neurodegenerative and mitochondrial diseases, where ATP production and redox balance are impaired.
Broader Impact
Drug Development:
The structural elucidation of MPC provides a molecular framework for designing selective modulators, setting the stage for new classes of metabolic drugs.
Precision Medicine:
Understanding individual differences in MPC structure/function may lead to personalized metabolic therapies tailored to genetic or disease-specific metabolic profiles.
Synthetic Biology & Bioenergetics:
The detailed MPC model can inform the engineering of customized metabolic pathways, supporting advances in synthetic biology, cell therapies, and biotechnology.
Keynote Speech: Targeting Mitochondrial Channels: Update and Strategies
Keynote Speech: Targeting Mitochondrial Channels: Update and Strategies
In his keynote speech, Prof. Szewczyk will provide the latest insights into the role of mitochondrial channels in cellular function and disease. He will discuss recent advancements and strategic approaches for targeting these channels, highlighting their potential in therapeutic interventions.
About Adam Szewczyk
Adam Szewczyk is a Professor of Biochemistry and former director of the Nencki Institute of Experimental Biology (Polish Academy of Sciences) in Poland, and since 2022, he has served as the President of the Polish Biochemical Society. He completed his chemical studies at Warsaw University and his postdoctoral fellowship at Bern University (Switzerland), Institute of Cellular and Molecular Pharmacology-Nice University (France), and at Johns Hopkins University, Baltimore, MD. He is the Head of the Laboratory of Intracellular Ion Channels at Nencki Institute.
His research is focused on the role of ion channels on mitochondrial function and intracellular ion channels pharmacology and biophysical properties of mitochondrial potassium channels.
World Mitochondria Society
Annual World Congress on Targeting Mitochondria
October 22-24, 2025 - Berlin, Germany
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Development of Mitochondria-Based Therapeutic Strategies for Disease Treatment
We are pleased to announce that Prof. Kosuke Kusamori from Tokyo University of Science, Japan, will be presenting his pioneering research on "Development of Mitochondria-Based Therapeutic Strategies for Disease Treatment."
Summary
In recent years, the application of mitochondria isolated from cells for disease treatment has gained increasing attention, with their efficacy demonstrated in several diseases. However, the functions and characteristics of isolated mitochondria remain largely unknown, and their kinetics after administration into the body have yet to be fully elucidated.
Prof. Kusamori has been investigating the physical properties and in vivo kinetics of isolated mitochondria. In this talk, Prof. Kusamori will present his research on mitochondria-based therapeutic strategies aimed at advancing mitochondrial therapeutics.
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Annual World Congress on Targeting Mitochondria
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How Mitochondria Organize Their Powerhouse Machinery for Optimal Performance
We are pleased to announce that Dr. Florent Waltz from the University of Basel, Switzerland, will be presenting at the Targeting Mitochondria 2025 congress in Berlin, Germany, on October 22-24, 2025.
Dr. Waltz will share insights from his groundbreaking research on "How Mitochondria Organize Their Powerhouse Machinery for Optimal Performance" with a special focus on photosynthetic organisms.
Key Highlights:
- In Situ Visualization: Researchers employed advanced imaging techniques to observe the mitochondrial respiratory chain within intact cells, providing a detailed view of its native architecture.
- Respiratory Supercomplexes: The study offers insights into how respiratory complexes assemble into supercomplexes, which are crucial for efficient electron transport and energy production in cells.
- Functional Implications: Understanding the organization of these supercomplexes sheds light on their role in cellular metabolism and energy conversion, potentially informing research into mitochondrial-related diseases.
Perspective:
- Challenging previous assumptions: The findings challenge long-standing models that assumed a more fluid, random distribution of respiratory chain components in mitochondrial membranes.
- Biological relevance: By analyzing structures in situ, this study underscores the importance of studying macromolecular organization in native cellular contexts, rather than relying only on purified proteins.
- Broader implications: These insights are critical not only for basic mitochondrial biology but also for understanding mitochondrial dysfunction in aging, neurodegenerative diseases, and metabolic disorders.
- New model for mitochondrial function: This study supports a model in which the geometrical and biochemical compartmentalization within cristae contributes significantly to the efficiency of oxidative phosphorylation.
These findings enhance our comprehension of mitochondrial function and may have implications for addressing metabolic disorders linked to mitochondrial dysfunction.
About the Speaker:
Dr. Florent Waltz leads research at the University of Basel focusing on mitochondrial biology and evolution in photosynthetic organisms, particularly micro-algae. His laboratory employs state-of-the-art imaging technologies to reveal the intricate details of how these essential organelles function and adapt.
Prof. Yosuke Togashi to Present Groundbreaking Insights on Mitochondrial Transfer and Immune Evasion in Cancer
Prof. Togashi’s upcoming lecture follows his landmark publication in Nature (February 2025), where his team uncovered a novel immune evasion mechanism in cancer: the direct transfer of mutated mitochondria from cancer cells to tumor-infiltrating T cells (TILs).
Why Prof. Togashi’s Contribution Is Strategic
- Discovery of Mitochondrial Transfer as an Immune Evasion Mechanism
Prof. Togashi and colleagues demonstrated that cancer cells transfer mutated mitochondria to tumor-infiltrating T cells (TILs), leading to functional impairment of the immune response. - Homoplasmic Replacement in T Cells
This transfer causes replacement of healthy T cell mitochondria with cancer-derived, mutation-bearing mitochondria—effectively sabotaging T cell metabolism, memory formation, and anti-tumor function. - Inhibition of Mitophagy via USP30
He uncovered that mitophagy-inhibitory molecules, especially USP30, are transferred along with the mitochondria, preventing the degradation of dysfunctional mitochondria in T cells. - Impact on Immunotherapy Resistance
The study linked mitochondrial transfer to poor response to immune checkpoint inhibitors (ICIs) in melanoma and lung cancer patients, providing a novel biomarker and therapeutic target to overcome resistance. - Therapeutic Reversibility
Blocking mitochondrial transfer or inhibiting USP30 restored T cell function, paving the way for new combinatory approaches to boost immunotherapy efficacy.
A New Era in Cancer Immunometabolism
Prof. Togashi’s insights underscore the critical need to rethink cancer metabolism and immune resistance at the mitochondrial level. His work bridges cancer biology, immunology, and mitochondrial medicine—a core mission of the Targeting Mitochondria community.
Fighting Fire with Fire: Boosting T Cell Therapy by Intercellular Mitochondrial Transfer
It is a great pleasure to announce that Prof. Luca Gattinoni, Leibniz Institute for Immunotherapy, Germany, will join Targeting Mitochondria 2025 as a major speaker.
Presentation Title: Fighting Fire with Fire: Boosting T Cell Therapy by Intercellular Mitochondrial Transfer.
Key Points
- Mitochondrial loss and dysfunction drive T cell exhaustion, representing major barriers to successful T cell-based immunotherapies.
- Bone marrow stromal cells (BMSCs) form nanotubular connections with T cells, enabling mitochondrial transfer into T cells.
- Transferred mitochondria enhance T cell mitochondrial mass and metabolic fitness.
- Mitochondria-boosted T cells exhibit resistance to exhaustion and demonstrate superior antitumor activity.
World Mitochondria Society
Annual World Congress on Targeting Mitochondria
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Mitochondria and Creatine: Perspectives and Strategies
Dr. David Rizo Roca, Karolisnka Institutet, Sweden, will join the 16th World Congress on Targeting Mitochondria 2025 to tal about "Mitochondria and Creatine: Perspectives and Strategies".
Summary
Creatine supplementation is widely used for its benefits in muscle performance and energy metabolism. However, recent research has linked elevated creatine levels with a higher risk of type 2 diabetes.
This talk will explore the relationship between creatine metabolism, insulin resistance, and mitochondrial function, highlighting key findings from the latest research.
World Mitochondria Society
Annual World Congress on Targeting Mitochondria
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Brain Organoids for drug discovery of Mitochondrial Neurological Disorders
It is a great pleasure to announce that Prof. Alessandro Prigione, active member or WMS scientific committee, Heinrich Heine University, Germany, will join Targeting Mitochondria 2025 as a major speaker.
Presentation Title: Brain Organoids for drug discovery of Mitochondrial Neurological Disorders.
Summary
Brain organoids are three dimensional structures that can be generated from pluripotent stem cells to model human neurodevelopemt.
In this talk, Prof. Prigione will describe how we can harness brain organoids to identify molecular targets and possible disease-modifying interventions for currently incurable mitochondrial and neurological diseases such as Huntington´s disease and Leigh syndrome.
Join Targeting Mitochondria 2025 to learn more about Prof. Prigione's interesting findings. You can submit a related abstract here.
World Mitochondria Society
Annual World Congress on Targeting Mitochondria
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New Workshop on Mitochondrial Transfer and Transplantation: From Bench to Bedside
The World Mitochondria Society is pleased to announce a hands-on workshop, “Mitochondrial Transfer and Transplantation: From Bench to Bedside,” led by Dr. Andrés Caicedo, a prominent faculty member at the Universidad San Francisco de Quito (USFQ) School of Medicine, Ecuador, and Vice President for South and Central America of the International Society for Cell & Gene Therapy (ISCT).
This workshop will be held on October 22, 2025, at the DoubleTree by Hilton Berlin, as part of the pre-congress activities for the Targeting Mitochondria 2025 Congress.
Building on the success of the traditional “Mitochondria Evaluation” Workshop conducted by Dr. Naig Gueguen, it offers an immersive and practical learning experience.
Participants will explore advanced techniques in studying horizontal mitochondrial transfer (HMT) between cells, the isolation and artificial transfer of mitochondria to other cells ex vivo, their reimplantation into hosts, and the methodologies behind mitochondrial transplantation. The session will also address therapeutic applications, key challenges, and future prospects in this innovative field.
Key points to Be Covered
1. How it works – Understanding mitochondrial transfer and transplantation.
2. Barriers and pitfalls to avoid – Challenges to navigate during the process.
3. Challenges and future perspectives – Emerging applications, issues and potential solutions.
4. Methods and tools – Supporting materials provided in the workshop book.
Supporting Material
1. Recorded Demonstrations – Key techniques for mitochondrial isolation and transplantation.
2. Case Studies and Applications – Real-world examples showcasing successful implementations in research and clinical settings.
3. Emerging Technologies – Innovative tools and methods reshaping the field of mitochondrial transfer and transplantation.
4. Interactive Q&A – A dedicated session for addressing specific questions, sharing challenges, and proposing solutions with expert input.
5. Workshop Book – Tangible takeaways for participants, including summaries of techniques, resources, publications and a certificate of participation.
6. Follow-up Webinars – Post-meeting sessions to track progress and share updates, fostering a sense of community within the WMS.
Abstracts of Targeting Mitochondria 2024 are Published in the "Journal of Mitochondria, Plastids and Endosymbiosis"
It is a great pleasure to announce that all abstracts accepted at Targeting Mitochondria 2024 are now published in the journal by Taylor & Francis "Journal of Mitochondria, Plastids and Endosymbiosis" - with a DOI !
This publication serves as a lasting record of the innovative research and findings shared during the congress. All abstracts are accessible under a single DOI, ensuring easy reference and seamless access for researchers and professionals around the world.
Access the abstracts: https://doi.org/10.1080/28347056.2024.2422726.
More Articles...
- WMS Best Short Oral Presentation: GPCR Agonist CAP-1902 Enhances Mitophagy to Improve Mitochondrial Function in Complex III Deficiency
- Exploring Mitochondria-Containing Extracellular Vesicles in Alzheimer’s Disease Wins WMS Best Poster Presentation (2)
- Gallbladder Mitochondrial Function as a Predictor of Liver Transplantation Outcomes Wins WMS Best Poster (2)
- Nominations for Best Mitochondria Image 2024
